Protodioscin Induces Apoptosis Through ROS-Mediated Endoplasmic Reticulum Stress via the JNK/p38 Activation Pathways in Human Cervical Cancer Cells | Zendy

ChiaLiang Lin | Zendy; Chien-Hsing Lee | Zendy; ChienMin Chen | Zendy; ChunWen Cheng | Zendy; PeiNi Chen | Zendy; TsungHo Ying | Zendy; YiHsien Hsieh | Zendy

Open Access

Protodioscin Induces Apoptosis Through ROS-Mediated Endoplasmic Reticulum Stress via the JNK/p38 Activation Pathways in Human Cervical Cancer Cells

Author(s) -

ChiaLiang Lin,

Chien-Hsing Lee,

ChienMin Chen,

ChunWen Cheng,

PeiNi Chen,

TsungHo Ying,

YiHsien Hsieh

Publication year - 2018

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000488433

Subject(s) - unfolded protein response , apoptosis , endoplasmic reticulum , viability assay , p38 mitogen activated protein kinases , mtt assay , chop , microbiology and biotechnology , programmed cell death , cancer cell , chemistry , cancer research , signal transduction , biology , mapk/erk pathway , cancer , biochemistry , genetics

Protodioscin (PD) is a steroidal saponin with anti-cancer effects on a number of cancer cells, but the anti-tumor effects and mechanism of action of PD on human cervical cancer cells is unclear.

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