Expressions and Regulatory Effects of P38/ERK/JNK Mapks in the Adipogenic Trans-Differentiation of C2C12 Myoblasts | Zendy

Renli Qi | Zendy; Hong Liu | Zendy; Qi Wang | Zendy; Jing Wang | Zendy; Feiyun Yang | Zendy; Dingbiao Long | Zendy; Jinxiu Huang | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Expressions and Regulatory Effects of P38/ERK/JNK Mapks in the Adipogenic Trans-Differentiation of C2C12 Myoblasts

Author(s) -

Renli Qi,

Hong Liu,

Qi Wang,

Jing Wang,

Feiyun Yang,

Dingbiao Long,

Jinxiu Huang

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000486169

Subject(s) - adipogenesis , c2c12 , p38 mitogen activated protein kinases , mapk/erk pathway , microbiology and biotechnology , cellular differentiation , kinase , myocyte , adipocyte , protein kinase a , chemistry , biology , endocrinology , myogenesis , adipose tissue , biochemistry , gene , mesenchymal stem cell

Myoblasts and muscle satellite cells have the potential to transdifferentiate into adipocytes or adipocyte-like cells. Previous studies suggest that mitogen-activated protein kinase (MAPK) is critical to adipogenic trans-differentiation of muscle cells. ERK1/2, P38 and JNK are three major MAPK family members; their activation and regulatory functions during adipogenic trans-differentiation of myoblasts are investigated.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research