MiR-106b-5p Promotes Proliferation and Inhibits Apoptosis by Regulating BTG3 in Non-Small Cell Lung Cancer | Zendy

Ke Wei | Zendy; Chunfeng Pan | Zendy; Guoliang Yao | Zendy; Bin Liu | Zendy; Teng Ma | Zendy; Yang Xia | Zendy; Wei Jiang | Zendy; Chen Liang | Zendy; Yijiang Chen | Zendy

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MiR-106b-5p Promotes Proliferation and Inhibits Apoptosis by Regulating BTG3 in Non-Small Cell Lung Cancer

Author(s) -

Ke Wei,

Chunfeng Pan,

Guoliang Yao,

Bin Liu,

Teng Ma,

Yang Xia,

Wei Jiang,

Chen Liang,

Yijiang Chen

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000485650

Subject(s) - carcinogenesis , cell growth , apoptosis , cell cycle , microrna , cancer research , biology , in vivo , cell cycle checkpoint , cell , flow cytometry , luciferase , cell culture , cancer , immunology , gene , transfection , biochemistry , genetics

MicroRNAs have been validated to play a crucial role in tumorigenesis of non-small cell lung cancer (NSCLC). Although miR-106b-5p has been reported to play a vital role in various malignancies the physiological function of miR-106b-5p in NSCLC still remain unknown. In this study, we investigated the role of miR-106b-5p in NSCLC.

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