Mir-21 Promotes Cardiac Fibrosis After Myocardial Infarction Via Targeting Smad7 | Zendy

Jinxia Yuan | Zendy; Hongtao Chen | Zendy; Dawei Ge | Zendy; Yu Xu | Zendy; Haihua Xu | Zendy; Yang Yang | Zendy; Ming Gu | Zendy; Yuhe Zhou | Zendy; Jingdong Zhu | Zendy; Ting Ge | Zendy; Qun Chen | Zendy; Yue Gao | Zendy; Yanqing Wang | Zendy; Xiaowei Li | Zendy; Y Zhao | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Mir-21 Promotes Cardiac Fibrosis After Myocardial Infarction Via Targeting Smad7

Author(s) -

Jinxia Yuan,

Hongtao Chen,

Dawei Ge,

Yu Xu,

Haihua Xu,

Yang Yang,

Ming Gu,

Yuhe Zhou,

Jingdong Zhu,

Ting Ge,

Qun Chen,

Yue Gao,

Yanqing Wang,

Xiaowei Li,

Y Zhao

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000479995

Subject(s) - in vivo , myocardial infarction , fibrosis , cardiac fibrosis , western blot , medicine , myocardial fibrosis , luciferase , reporter gene , microrna , cardiac function curve , transforming growth factor , cancer research , gene expression , cardiology , biology , heart failure , cell culture , transfection , gene , biochemistry , genetics , microbiology and biotechnology

Cardiac fibrosis after myocardial infarction (MI) has been identified as an important factor in the deterioration of heart function. Previous studies have demonstrated that miR-21 plays an important role in various pathophysiological processes in the heart. However, the role of miR-21 in fibrosis regulation after MI remains unclear.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research