Suppression of STAT3 Signaling by Δ9-Tetrahydrocannabinol (THC) Induces Trophoblast Dysfunction | Zendy

Xinwen Chang | Zendy; Yiding Bian | Zendy; Qizhi He | Zendy; Julei Yao | Zendy; Jingping Zhu | Zendy; Jinting Wu | Zendy; Kai Wang | Zendy; Tao Duan | Zendy

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Suppression of STAT3 Signaling by Δ9-Tetrahydrocannabinol (THC) Induces Trophoblast Dysfunction

Author(s) -

Xinwen Chang,

Yiding Bian,

Qizhi He,

Julei Yao,

Jingping Zhu,

Jinting Wu,

Kai Wang,

Tao Duan

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000477603

Subject(s) - trophoblast , cannabinoid receptor , cannabinoid , placenta , receptor , biology , stat3 , tetrahydrocannabinol , signal transduction , stat protein , andrology , medicine , endocrinology , fetus , microbiology and biotechnology , pharmacology , pregnancy , antagonist , genetics

Marijuana is a widely used illicit drug and its consumption during pregnancy has been associated with adverse reproductive outcomes. The purpose of this study was to determine the effects of chronic intake of Δ9-tetrahydrocannabinol (THC), the major component of marijuana, on trophoblast function, placental development, and birth outcomes.

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