Heat Acclimation Regulates the Autophagy-Lysosome Function to Protect Against Heat Stroke-Induced Brain Injury in Mice | Zendy

Junfeng Yi | Zendy; Genlin He | Zendy; Ju Yang | Zendy; Zhen Luo | Zendy; Xuesen Yang | Zendy; Xue Luo | Zendy

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Heat Acclimation Regulates the Autophagy-Lysosome Function to Protect Against Heat Stroke-Induced Brain Injury in Mice

Author(s) -

Junfeng Yi,

Genlin He,

Ju Yang,

Zhen Luo,

Xuesen Yang,

Xue Luo

Publication year - 2017

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000455979

Subject(s) - autophagy , lysosome , neuroprotection , microbiology and biotechnology , tunel assay , programmed cell death , chemistry , biology , apoptosis , pharmacology , biochemistry , enzyme

The mechanisms underlying the protective role of heat acclimation (HA) in heat stroke (HS)-induced brain injury are still unclear. The autophagy-lysosome pathway is known to pay an important role in protecting stressed or diseased cells from death. Nevertheless, whether autophagy and lysosomes are involved in HA-mediated neuroprotection following HS exposure remains unclear.

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