Inhibition of the JAK2/STAT3/SOSC1 Signaling Pathway Improves Secretion Function of Vascular Endothelial Cells in a Rat Model of Pregnancy-Induced Hypertension | Zendy

Jianying Luo | Zendy; Dan Fu | Zendy; Yaqin Wu | Zendy; Ying Gao | Zendy

Open Access

Inhibition of the JAK2/STAT3/SOSC1 Signaling Pathway Improves Secretion Function of Vascular Endothelial Cells in a Rat Model of Pregnancy-Induced Hypertension

Author(s) -

Jianying Luo,

Dan Fu,

Yaqin Wu,

Ying Gao

Publication year - 2016

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000452566

Subject(s) - medicine , endocrinology , nitric oxide , tumor necrosis factor alpha , preeclampsia , interleukin 6 , stat3 , radioimmunoassay , interleukin , thromboxane b2 , thromboxane , endothelin receptor , pregnancy , signal transduction , cytokine , chemistry , receptor , biology , platelet , genetics , biochemistry

The present study aimed to investigate the effects of the JAK2/STAT3/SOSC1 signaling pathway on the secretion function of vascular endothelial cells (VECs) in a rat model of pregnancy-induced hypertension (PIH).

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