Recurrence of Hyperoxaluria and Kidney Disease after Combined Intestine-Kidney Transplantation for Enteric Hyperoxaluria
Author(s) -
Burcin Ekser,
Richard S. Mangus,
Chandrashekhar A. Kubal,
Jonathan A. Fridell,
John A. Powelson,
Santosh Nagaraju,
Plamen Mihaylov,
Carrie L. Phillips,
Romil Saxena,
William C. Goggins
Publication year - 2016
Publication title -
american journal of nephrology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.394
H-Index - 85
eISSN - 1421-9670
pISSN - 0250-8095
DOI - 10.1159/000447785
Subject(s) - medicine , transplantation , kidney disease , gastroenterology , surgery , kidney transplantation , acute tubular necrosis , primary hyperoxaluria , short bowel syndrome , kidney , nephropathy , parenteral nutrition , endocrinology , diabetes mellitus
Enteric hyperoxaluria (EH) occurs with a rate of 5-24% in patients with inflammatory bowel disease, ileal resection and modern bariatric surgery. The excessive absorption of calcium oxalate causes chronic kidney disease (CKD) in patients with EH. In the literature, a single experience was reported in combined intestine-kidney transplantation (CIKTx) in patients with CKD due to EH.
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