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Ginsenoside Rh2 Suppresses Neovascularization in Xenograft Psoriasis Model
Author(s) -
Jing Zhou,
Yunlu Gao,
Xue-Mei Yi,
Yangfeng Ding
Publication year - 2015
Publication title -
cellular physiology and biochemistry
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.486
H-Index - 87
eISSN - 1421-9778
pISSN - 1015-8987
DOI - 10.1159/000430272
Subject(s) - acanthosis , medicine , psoriasis , neovascularization , papillomatosis , angiogenesis , cd31 , pathology , immunology , hyperkeratosis
Psoriasis is a common inflammatory skin disease of undetermined etiology and poor prognosis. The current therapies have focused on direct inhibition of local inflammation, e.g. through hormone treatments. However, neovascularization plays a critical role in the development of psoriasis but so far no therapies have been developed to suppress psoriasis-associated neovascularization.

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