MiR-142-3p Suppresses SOCS6 Expression and Promotes Cell Proliferation in Nasopharyngeal Carcinoma | Zendy

Xiaoming Qi | Zendy; Jianqiang Li | Zendy; Changbo Zhou | Zendy; Chunlei Lv | Zendy; Min Tian | Zendy

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MiR-142-3p Suppresses SOCS6 Expression and Promotes Cell Proliferation in Nasopharyngeal Carcinoma

Author(s) -

Xiaoming Qi,

Jianqiang Li,

Changbo Zhou,

Chunlei Lv,

Min Tian

Publication year - 2015

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000430147

Subject(s) - nasopharyngeal carcinoma , gene knockdown , microrna , cell growth , cancer research , socs3 , carcinogenesis , socs6 , biology , suppressor of cytokine signaling 1 , downregulation and upregulation , suppressor , cell , cell culture , signal transduction , microbiology and biotechnology , stat3 , gene , medicine , genetics , radiation therapy

An increasing number of studies show that microRNAs (miRNAs) play crucial roles in nasopharyngeal carcinoma (NPC) tumorigenesis. The aim of our study was to investigate the biological roles and mechanisms of miR-142-3p in NPC.

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