High T3, Low T4 Serum Levels in Mct8 Deficiency Are Not Caused by Increased Hepatic Conversion through Type I Deiodinase | Zendy

Eva K. Wirth | Zendy; Eddy Rijntjes | Zendy; Franziska Meyer | Zendy; Josef Köhrle | Zendy; Ulrich Schweizer | Zendy

Open Access

High T3, Low T4 Serum Levels in Mct8 Deficiency Are Not Caused by Increased Hepatic Conversion through Type I Deiodinase

Author(s) -

Eva K. Wirth,

Eddy Rijntjes,

Franziska Meyer,

Josef Köhrle,

Ulrich Schweizer

Publication year - 2015

Publication title -

european thyroid journal

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.23

H-Index - 10

eISSN - 2235-0802

pISSN - 2235-0640

DOI - 10.1159/000381021

Subject(s) - deiodinase , medicine , endocrinology , monocarboxylate transporter , thyroid , hormone , symporter , iodothyronine deiodinase , transporter , triiodothyronine , biology , gene , biochemistry

The Allan-Herndon-Dudley syndrome is a severe psychomotor retardation accompanied by specific changes in circulating thyroid hormone levels (high T3, low T4). These are caused by mutations in the thyroid hormone transmembrane transport protein monocarboxylate transporter 8 (MCT8).

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