Emodin Ameliorates High Glucose Induced-Podocyte Epithelial-Mesenchymal Transition In-Vitro and In-Vivo | Zendy

Tingfang Chen | Zendy; Li Zheng | Zendy; Wenzhen Xiao | Zendy; Dingkun Gui | Zendy; Xiaoxia Wang | Zendy; Niansong Wang | Zendy

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Emodin Ameliorates High Glucose Induced-Podocyte Epithelial-Mesenchymal Transition In-Vitro and In-Vivo

Author(s) -

Tingfang Chen,

Li Zheng,

Wenzhen Xiao,

Dingkun Gui,

Xiaoxia Wang,

Niansong Wang

Publication year - 2015

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000373963

Subject(s) - nephrin , podocyte , desmin , podocin , slit diaphragm , albuminuria , endocrinology , medicine , chemistry , in vivo , diabetic nephropathy , cancer research , kidney , vimentin , biology , immunohistochemistry , proteinuria , microbiology and biotechnology

Epithelial-to-mesenchymal transition (EMT) is a potential pathway leading to podocyte depletion and proteinuria in diabetic kidney disease (DKD). Here, we investigated the protective effects of Emodin (EMO) on high glucose (HG) induced-podocyte EMT in-vitro and in-vivo.

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