Tetranectin Knockout Mice Develop Features of Parkinson Disease | Zendy

Ersong Wang | Zendy; Xiaoping Zhang | Zendy; Hui-bin Yao | Zendy; Wang Gan | Zendy; Shiwen Chen | Zendy; Wenwei Gao | Zendy; Haijun Yao | Zendy; Yirui Sun | Zendy; Caihua Xi | Zendy; Yao-dong Ji | Zendy

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Tetranectin Knockout Mice Develop Features of Parkinson Disease

Author(s) -

Ersong Wang,

Xiaoping Zhang,

Hui-bin Yao,

Wang Gan,

Shiwen Chen,

Wenwei Gao,

Haijun Yao,

Yirui Sun,

Caihua Xi,

Yao-dong Ji

Publication year - 2014

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000362998

Subject(s) - pars compacta , substantia nigra , dopaminergic , neuroscience , striatum , dopamine , neuroprotection , parkinson's disease , synucleinopathies , midbrain , mptp , parkinsonism , pathogenesis , biology , knockout mouse , alpha synuclein , medicine , pathology , disease , central nervous system , receptor

Aggregation of insoluble α-synuclein to form Lewy bodies (LBs) may contribute to the selective loss of midbrain dopaminergic neurons in Parkinson disease (PD). Lack of robust animal models has impeded elucidation of the molecular mechanisms of LB formation and other critical aspects of PD pathogenesis.

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