The Involvement of Phosphatase and Tensin Homolog Deleted on Chromosome Ten (PTEN) in the Regulation of Inflammation Following Coronary Microembolization | Zendy

Jiangyou Wang | Zendy; Lang Li | Zendy; Qiang Su | Zendy; You Zhou | Zendy; Han Chen | Zendy; Guo-Tian Ma | Zendy; Tao Liu | Zendy; Zhongli Tang | Zendy; Yangchun Liu | Zendy

Open Access

The Involvement of Phosphatase and Tensin Homolog Deleted on Chromosome Ten (PTEN) in the Regulation of Inflammation Following Coronary Microembolization

Author(s) -

Jiangyou Wang,

Lang Li,

Qiang Su,

You Zhou,

Han Chen,

Guo-Tian Ma,

Tao Liu,

Zhongli Tang,

Yangchun Liu

Publication year - 2014

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000362973

Subject(s) - pten , tensin , phosphatase , inflammation , downregulation and upregulation , cancer research , ejection fraction , medicine , biology , pathology , heart failure , phosphorylation , pi3k/akt/mtor pathway , microbiology and biotechnology , gene , signal transduction , genetics

Growing evidence shows that phosphatase and tensin homolog deleted on chromosome ten (PTEN) is involved in regulating inflammation in different pathological conditions. Therefore, we hypothesized that the upregulation of PTEN correlates with the impairment of cardiac function in swine following coronary microembolization (CME).

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