Neuronostatin Attenuates Myocardial Contractile Function through Inhibition of Sarcoplasmic Reticulum Ca2+-ATPase in Murine Heart | Zendy

Xiaoling Zhu | Zendy; Nan Hu | Zendy; Xiyao Chen | Zendy; Miaozhang Zhu | Zendy; Hailong Dong | Zendy; Xihui Xu | Zendy; Fuling Luo | Zendy; Yinan Hua | Zendy; Sreejayan Nair | Zendy; Willis K. Samson | Zendy; Lize Xiong | Zendy; Jun Ren | Zendy

Open Access

Neuronostatin Attenuates Myocardial Contractile Function through Inhibition of Sarcoplasmic Reticulum Ca²⁺-ATPase in Murine Heart

Author(s) -

Xiaoling Zhu,

Nan Hu,

Xiyao Chen,

Miaozhang Zhu,

Hailong Dong,

Xihui Xu,

Fuling Luo,

Yinan Hua,

Sreejayan Nair,

Willis K. Samson,

Lize Xiong,

Jun Ren

Publication year - 2014

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000362969

Subject(s) - phospholamban , serca , medicine , endocrinology , intracellular , cardiac function curve , endoplasmic reticulum , contractility , homeostasis , phosphorylation , ampk , sarcolemma , atpase , chemistry , myocyte , biology , heart failure , protein kinase a , microbiology and biotechnology , biochemistry , enzyme

Neuronostatin, derived from the somatostatin preprohormone, was recently identified to be produced by several tissues exerting a role in cardiovascular regulation and metabolism. Nonetheless, the precise mechanism behind neuronostatin-elicited myocardial responses remains elusive.

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