Autophagy Protects Against Senescence and Apoptosis via the RAS-Mitochondria in High-Glucose-Induced Endothelial Cells | Zendy

Fei Chen | Zendy; Bin Chen | Zendy; Fen-Qiang Xiao | Zendy; Yutao Wu | Zendy; Ri-Hong Wang | Zendy; Zewei Sun | Zendy; Guosheng Fu | Zendy; Yun Mou | Zendy; Wu Tao | Zendy; Xiaosheng Hu | Zendy; ShenJiang Hu | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Autophagy Protects Against Senescence and Apoptosis via the RAS-Mitochondria in High-Glucose-Induced Endothelial Cells

Author(s) -

Fei Chen,

Bin Chen,

Fen-Qiang Xiao,

Yutao Wu,

Ri-Hong Wang,

Zewei Sun,

Guosheng Fu,

Yun Mou,

Wu Tao,

Xiaosheng Hu,

ShenJiang Hu

Publication year - 2014

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000358676

Subject(s) - autophagy , apoptosis , senescence , microbiology and biotechnology , mitochondrion , angiotensin ii , oxidative stress , endothelial dysfunction , chemistry , homeostasis , biology , endocrinology , medicine , receptor , biochemistry

Autophagy is an important process in the pathogenesis of diabetes and plays a critical role in maintaining cellular homeostasis. However, the autophagic response and its mechanism in diabetic vascular endothelium remain unclear.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research