Non-Lethal Levels of Oxidative Stress in Response to Short-Term Intermittent Hypoxia Enhance Ca2+Handling in Neonatal Rat Cardiomyocytes | Zendy

Tsung-I Chen | Zendy; YuCheng Hsu | Zendy; ChihFeng Lien | Zendy; Jianhong Lin | Zendy; HungWen Chiu | Zendy; KunTa Yang | Zendy

Open Access

Non-Lethal Levels of Oxidative Stress in Response to Short-Term Intermittent Hypoxia Enhance Ca²⁺Handling in Neonatal Rat Cardiomyocytes

Author(s) -

Tsung-I Chen,

YuCheng Hsu,

ChihFeng Lien,

Jianhong Lin,

HungWen Chiu,

KunTa Yang

Publication year - 2014

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000358631

Subject(s) - ryanodine receptor , cytosol , extracellular , protein kinase c , chemistry , oxidative stress , intracellular , endoplasmic reticulum , calcium , myocyte , microbiology and biotechnology , medicine , endocrinology , biology , biochemistry , kinase , enzyme , organic chemistry

Intermittent hypoxia (IH) may exert pre-conditioning-like cardioprotective effects and alter Ca(2+) regulation; however, the exact mechanism of these effects remains unclear. Thus, we examined Ca(2+)-handling mechanisms induced by IH in rat neonatal cardiomyocytes.

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