Apolipoprotein E-Deficient Mice Are Susceptible to the Development of Acute Lung Injury | Zendy

Cory Yamashita | Zendy; Michael B. Fessler | Zendy; Lakshman Vasanthamohan | Zendy; Joanne Lac | Zendy; Jennifer H. Madenspacher | Zendy; Lynda McCaig | Zendy; LiJuan Yao | Zendy; Lefeng Wang | Zendy; Valeria Puntorieri | Zendy; Sanjay Mehta | Zendy; Jim F. Lewis | Zendy; Ruud A. W. Veldhuizen | Zendy

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Apolipoprotein E-Deficient Mice Are Susceptible to the Development of Acute Lung Injury

Author(s) -

Cory Yamashita,

Michael B. Fessler,

Lakshman Vasanthamohan,

Joanne Lac,

Jennifer H. Madenspacher,

Lynda McCaig,

LiJuan Yao,

Lefeng Wang,

Valeria Puntorieri,

Sanjay Mehta,

Jim F. Lewis,

Ruud A. W. Veldhuizen

Publication year - 2014

Publication title -

respiration

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.264

H-Index - 81

eISSN - 1423-0356

pISSN - 0025-7931

DOI - 10.1159/000358438

Subject(s) - apolipoprotein e , medicine , inflammation , lung , vascular permeability , apolipoprotein b , in vivo , immunology , cytokine , endocrinology , cholesterol , biology , disease , microbiology and biotechnology

Apolipoprotein E (apoE) has been shown to play a pivotal role in the development of cardiovascular disease, attributable to its function in lipid trafficking and immune modulating properties; however, its role in modulating inflammation in the setting of acute lung injury (ALI) is unknown.

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