NOD2 Deficiency Protects against Cardiac Remodeling after Myocardial Infarction in Mice | Zendy

Xiang Li | Zendy; Fengli Li | Zendy; Yongli Chu | Zendy; Xiaojie Wang | Zendy; Hongyu Zhang | Zendy; Yanyan Hu | Zendy; Yan Zhang | Zendy; Ziying Wang | Zendy; Xinbing Wei | Zendy; Wencheng Jian | Zendy; Xiumei Zhang | Zendy; Fan Yi | Zendy

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NOD2 Deficiency Protects against Cardiac Remodeling after Myocardial Infarction in Mice

Author(s) -

Xiang Li,

Fengli Li,

Yongli Chu,

Xiaojie Wang,

Hongyu Zhang,

Yanyan Hu,

Yan Zhang,

Ziying Wang,

Xinbing Wei,

Wencheng Jian,

Xiumei Zhang,

Fan Yi

Publication year - 2013

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000356618

Subject(s) - inflammation , proinflammatory cytokine , nod2 , medicine , signal transduction , innate immune system , immunology , immune system , biology , microbiology and biotechnology

Although the pathogenesis of myocardial infarction (MI) is multifactorial, activation of innate immune system to induce inflammation has emerged as a key pathophysiological process in MI. NOD2, one member of the NOD-like receptor (NLR) family, plays an important role in the innate immune response. This study was to examine the role of NOD2 during MI.

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