ART1 Silencing Enhances Apoptosis of Mouse CT26 Cells via the PI3K/Akt/NF-&#954;B Pathway | Zendy

Ming Xiao | Zendy; Yi Tang | Zendy; Yalan Wang | Zendy; Lian Yang | Zendy; Xian Li | Zendy; Jing Kuang | Zendy; Guang-Lin Song | Zendy

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ART1 Silencing Enhances Apoptosis of Mouse CT26 Cells via the PI3K/Akt/NF-κB Pathway

Author(s) -

Ming Xiao,

Yi Tang,

Yalan Wang,

Lian Yang,

Xian Li,

Jing Kuang,

Guang-Lin Song

Publication year - 2013

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000356595

Subject(s) - apoptosis , small hairpin rna , flow cytometry , protein kinase b , cancer research , gene silencing , gene knockdown , pi3k/akt/mtor pathway , biology , chemistry , microbiology and biotechnology , biochemistry , gene

Colorectal carcinoma is one of the most common cancers world-wide, with high morbidity and mortality rates. Arginine ADP-ribosyltransferase 1(ART1) is an important ecto-ADP-ribose transferase and has been proven to be intimately involved in a number of biological processes. However, the influence of ART1 on survival and apoptosis of colorectal carcinoma cells and the potential mechanism of action of ART1 remain uncharacterized.

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