Arctigenin, a Potential Anti-Arrhythmic Agent, Inhibits Aconitine-Induced Arrhythmia by Regulating Multi-Ion Channels | Zendy

Zhenying Zhao | Zendy; Yongqiang Yin | Zendy; Hong Wu | Zendy; Min Jiang | Zendy; Lou Jian-shi | Zendy; Gang Bai | Zendy; Guo-An Luo | Zendy

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Arctigenin, a Potential Anti-Arrhythmic Agent, Inhibits Aconitine-Induced Arrhythmia by Regulating Multi-Ion Channels

Author(s) -

Zhenying Zhao,

Yongqiang Yin,

Hong Wu,

Min Jiang,

Lou Jian-shi,

Gang Bai,

Guo-An Luo

Publication year - 2013

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000354532

Subject(s) - aconitine , chemistry , pharmacology , in vivo , ion channel , anti arrhythmia agents , sodium channel , potassium channel , context (archaeology) , biophysics , sodium , medicine , receptor , biochemistry , biology , atrial fibrillation , paleontology , organic chemistry , microbiology and biotechnology

Arctigenin possesses biological activities, but its underlying mechanisms at the cellular and ion channel levels are not completely understood. Therefore, the present study was designed to identify the anti-arrhythmia effect of arctigenin in vivo, as well as its cellular targets and mechanisms.

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