Acetylcholine Attenuates Hypoxia/ Reoxygenation-Induced Mitochondrial and Cytosolic ROS Formation in H9c2 Cells via M2 Acetylcholine Receptor | Zendy

Yi Miao | Zendy; Jun Zhou | Zendy; Mei Zhao | Zendy; Jinjun Liu | Zendy; Lei Sun | Zendy; XiaoJiang Yu | Zendy; Xijing He | Zendy; Xiaoyue Pan | Zendy; Weijin Zang | Zendy

Open Access

Acetylcholine Attenuates Hypoxia/ Reoxygenation-Induced Mitochondrial and Cytosolic ROS Formation in H9c2 Cells via M2 Acetylcholine Receptor

Author(s) -

Yi Miao,

Jun Zhou,

Mei Zhao,

Jinjun Liu,

Lei Sun,

XiaoJiang Yu,

Xijing He,

Xiaoyue Pan,

Weijin Zang

Publication year - 2013

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000343360

Subject(s) - mitochondrial ros , reactive oxygen species , oxidative stress , acetylcholine , chemistry , xanthine oxidase , nadph oxidase , mitochondrion , pharmacology , biology , biochemistry , enzyme

The anti-infammatory and cardioprotective effect of acetylcholine (ACh) has been reported; nevertheless, whether and how ACh exhibits an antioxidant property against ischemia/reperfusion (I/R)-induced oxidative stress remains obscure.

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