Acute and Chronic Cardioprotection by the Enkephalin Analogue, Eribis Peptide 94, is Mediated via Activation of Nitric Oxide Synthase and Adenosine Triphosphate-Regulated Potassium Channels | Zendy

Garrett J. Gross | Zendy; Anna Hsu | Zendy; Kasem Nithipatikom | Zendy; Adam W. Pfeiffer | Zendy; Irina Bobrova | Zendy; Erik Bissessar | Zendy

Open Access

Acute and Chronic Cardioprotection by the Enkephalin Analogue, Eribis Peptide 94, is Mediated via Activation of Nitric Oxide Synthase and Adenosine Triphosphate-Regulated Potassium Channels

Author(s) -

Garrett J. Gross,

Anna Hsu,

Kasem Nithipatikom,

Adam W. Pfeiffer,

Irina Bobrova,

Erik Bissessar

Publication year - 2012

Publication title -

pharmacology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.51

H-Index - 59

eISSN - 1423-0313

pISSN - 0031-7012

DOI - 10.1159/000340058

Subject(s) - cardioprotection , nitric oxide synthase , nitric oxide , pharmacology , potassium channel , chemistry , enkephalin , adenosine , medicine , ischemia , receptor , opioid

Eribis peptide 94 (EP 94) is a new enkephalin derivative which potently binds to the µ- and δ-opioid receptor. In this study, we determined the effects of EP 94 and potential mechanism(s) involved in cardioprotection of the rat heart.

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