Integrin-Dependent Akt1 Activation Regulates PGC-1 Expression and Fatty Acid Oxidation | Zendy

Craig C. Beeson | Zendy; Gyda C. Beeson | Zendy; Haley Buff | Zendy; Juanita Eldridge | Zendy; Aiguo Zhang | Zendy; Arun Seth | Zendy; Marina Demcheva | Zendy; John N. Vournakis | Zendy; Robin C. MuiseHelmericks | Zendy

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Integrin-Dependent Akt1 Activation Regulates PGC-1 Expression and Fatty Acid Oxidation

Author(s) -

Craig C. Beeson,

Gyda C. Beeson,

Haley Buff,

Juanita Eldridge,

Aiguo Zhang,

Arun Seth,

Marina Demcheva,

John N. Vournakis,

Robin C. MuiseHelmericks

Publication year - 2012

Publication title -

journal of vascular research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.58

H-Index - 74

eISSN - 1423-0135

pISSN - 1018-1172

DOI - 10.1159/000332326

Subject(s) - pdk4 , carnitine , beta oxidation , chemistry , microbiology and biotechnology , fatty acid , biochemistry , peroxisome , receptor , biology , pyruvate dehydrogenase complex , enzyme

Poly-N-acetyl glucosamine nanofibers derived from a marine diatom have been used to increase cutaneous wound healing. These nanofibers exert their activity by specifically activating integrins, which makes them a useful tool for dissecting integrin-mediated pathways. We have shown that short-fiber poly-N-acetyl glucosamine nanofiber (sNAG) treatment of endothelial cells results in increased cell motility and metabolic rate in the absence of increased cell proliferation.

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