Targeting Mitochondria by Olesoxime or Complement 1q Binding Protein as a Novel Management for Autism: A Hypothesis | Zendy

Ahmad Ghanizadeh | Zendy

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Targeting Mitochondria by Olesoxime or Complement 1q Binding Protein as a Novel Management for Autism: A Hypothesis

Author(s) -

Ahmad Ghanizadeh

Publication year - 2011

Publication title -

molecular syndromology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.609

H-Index - 36

eISSN - 1661-8777

pISSN - 1661-8769

DOI - 10.1159/000332202

Subject(s) - autism , oxidative stress , mitochondrion , excitotoxicity , complement (music) , complement system , chemistry , microbiology and biotechnology , biology , medicine , biochemistry , immunology , glutamate receptor , antibody , receptor , psychiatry , mutant , gene , complementation

Excitotoxicity, oxidative stress, and mitochondrial dysfunction are associated with autism. Considering the preventive role of complement 1q binding protein or olesoxime for the opening of mitochondrial permeability transition pore mediated by oxidative stress, it is hypothesized that complement 1q binding protein or olesoxime may improve some symptoms of autism.

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