1α,25-Dihydroxycholecalciferol Induces Nitric Oxide Production in Cultured Endothelial Cells | Zendy

Claudio Molinari | Zendy; Francesca Uberti | Zendy; Elena Grossini | Zendy; Giovanni Vacca | Zendy; Stefano Carda | Zendy; Marco Invernizzi | Zendy; Carlo Cisari | Zendy

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1α,25-Dihydroxycholecalciferol Induces Nitric Oxide Production in Cultured Endothelial Cells

Author(s) -

Claudio Molinari,

Francesca Uberti,

Elena Grossini,

Giovanni Vacca,

Stefano Carda,

Marco Invernizzi,

Carlo Cisari

Publication year - 2011

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000330075

Subject(s) - enos , wortmannin , calcitriol receptor , p38 mitogen activated protein kinases , nitric oxide , mapk/erk pathway , protein kinase b , medicine , umbilical vein , endocrinology , chemistry , microbiology and biotechnology , phosphorylation , stimulation , receptor , biology , nitric oxide synthase , biochemistry , in vitro

Recently, 1α,25-dihydroxycholecalciferol (vitD) has received increasing interest for its effects on many tissues and organs other than bone. A number of experimental studies have shown that vitD may have an important role in modifying risk for cardiovascular disease.

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