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We have developed an in vitro model by organ culture of rat mesenteric arteries to imitate vascular smooth muscle cell (VSMC) receptor changes in cardiovascular disease. By using this model, alteration of VSMC thromboxane A(2) (TP) receptors was studied.

Transcriptional Down-Regulation of Thromboxane A<sub>2</sub> Receptor Expression via Activation of MAPK ERK1/2, p38/NF-κB Pathways