Exogenous and Endogenous Nitric Oxide Donors Improve Post-Ischemic Tissue Oxygenation in Early Pancreatic Ischemia/Reperfusion Injury in the Rat | Zendy

Robert Obermaier | Zendy; Ernst von Dobschuetz | Zendy; A. Benthues | Zendy; N. Ansorge | Zendy; W. Schareck | Zendy; U. T. Hopt | Zendy; Stefan Benz | Zendy

Open Access

Exogenous and Endogenous Nitric Oxide Donors Improve Post-Ischemic Tissue Oxygenation in Early Pancreatic Ischemia/Reperfusion Injury in the Rat

Author(s) -

Robert Obermaier,

Ernst von Dobschuetz,

A. Benthues,

N. Ansorge,

W. Schareck,

U. T. Hopt,

Stefan Benz

Publication year - 2004

Publication title -

european surgical research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.658

H-Index - 46

eISSN - 1421-9921

pISSN - 0014-312X

DOI - 10.1159/000078856

Subject(s) - sodium nitroprusside , nitric oxide , reperfusion injury , ischemia , endocrinology , edema , ischemic preconditioning , medicine , chemistry

In pancreatic ischemia/reperfusion (IR) injury (IRI) the role of nitric oxide (NO) is not completely understood. Using a rat model of normothermic in situ IRI, the effect of endogenous and exogenous NO donors on post-ischemic tissue oxygenation and tissue damage was investigated.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research