Fanconi and Inappropriate Secretion of Antidiuretic Hormone Syndromes Secondary to Venlafaxine Therapy
Author(s) -
Hassane Izzedine,
Vincent LaunayVacher,
Magali Ciroldi,
L. Fardet,
Gilbert Deray
Publication year - 2002
Publication title -
the nephron journals/nephron journals
Language(s) - English
Resource type - Journals
eISSN - 2235-3186
pISSN - 1660-8151
DOI - 10.1159/000065455
Subject(s) - medicine , endocrinology , anion gap , creatinine , urine osmolality , hypokalemia , hyponatremia , aldosterone , renal function , metabolic acidosis
Accessible online at: www.karger.com/journals/nef Venlafaxine, a 2-phenyl-2 (1-hydroxycycloalkyl) ethylamine derivate, is an inhibitor of both serotonin and norepinephrine re-uptake and was termed a ‘dual uptake inhibitor’ [1]. We report on a venlafaxine-treated patient who developed Fanconi’s syndrome (FS) and hyponatremia secondary to inappropriate antidiuretic hormone secretion syndrome (SIADH).
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