Metabolic Syndrome in Rheumatoid Arthritis | Zendy

Iván FerrazAmaro | Zendy; Carlos GonzálezJuanatey | Zendy; Raquel LópezMejías | Zendy; Leyre RianchoZarrabeitia | Zendy; Miguel Á. GonzálezGay | Zendy

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Metabolic Syndrome in Rheumatoid Arthritis

Author(s) -

Iván FerrazAmaro,

Carlos GonzálezJuanatey,

Raquel LópezMejías,

Leyre RianchoZarrabeitia,

Miguel Á. GonzálezGay

Publication year - 2013

Publication title -

mediators of inflammation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.37

H-Index - 97

eISSN - 1466-1861

pISSN - 0962-9351

DOI - 10.1155/2013/710928

Subject(s) - rheumatoid arthritis , metabolic syndrome , medicine , arthritis , immunology , bioinformatics , biology , obesity

Insulin resistance is an essential feature of the metabolic syndrome that has been linked to rheumatoid arthritis (RA). Understanding how inflammation arising in one tissue affects the physiology and pathology of other organs remains an unanswered question with therapeutic implications for chronic conditions including obesity, diabetes mellitus, atherosclerosis, and RA. Adipokines may play a role in the development of atherogenesis in patients with RA. Biologic therapies, such as TNF- α antagonists, that block proinflammatory cytokines have beneficial effects on the insulin resistance that is often observed in patients with RA.

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