NETosis | Zendy

Miguel Antonio Mesa Navas | Zendy; Gloria Vásquez | Zendy

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NETosis

Author(s) -

Miguel Antonio Mesa Navas,

Gloria Vásquez

Publication year - 2013

Publication title -

autoimmune diseases

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.681

H-Index - 32

eISSN - 2090-0422

pISSN - 2090-0430

DOI - 10.1155/2013/651497

Subject(s) - neutrophil extracellular traps , sepsis , limiting , immune system , medicine , collateral damage , programmed cell death , immunology , autoimmune disease , microbiology and biotechnology , inflammation , biology , antibody , genetics , apoptosis , mechanical engineering , criminology , sociology , engineering

Neutrophils are the first line of defense of the immune system against infection. Among their weaponry, they have the ability to mix and extrude their DNA and bactericidal molecules creating NET-like structures in a unique type of cell death called NETosis. This process is important in order to control extracellular infections limiting collateral damage. Its aberrant function has been implicated in several human diseases including sepsis and autoimmune disease. The purpose of the present paper is to give a general introduction to this concept.

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