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The Effect of Endogenous Angiotensin II on Alveolar Fluid Clearance in Rats with Acute Lung Injury
Author(s) -
Jia Deng,
Daoxin Wang,
Wang Deng,
Changyi Li,
Jin Tong
Publication year - 2012
Publication title -
canadian respiratory journal
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.675
H-Index - 53
eISSN - 1916-7245
pISSN - 1198-2241
DOI - 10.1155/2012/951025
Subject(s) - medicine , epithelial sodium channel , angiotensin ii , pulmonary edema , lung , receptor , lipopolysaccharide , endocrinology , western blot , edema , endogeny , stimulation , renin–angiotensin system , pharmacology , sodium , blood pressure , chemistry , biochemistry , organic chemistry , gene
In acute lung injury (ALI), angiotensin II (Ang II) plays a vital role in the stimulation of pulmonary permeability edema formation through the angiotensin type 1 (AT1) receptor. The effect of Ang II on alveolar fluid clearance (AFC) in ALI remains unknown.

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