Traumatic Brain Injury, Microglia, and Beta Amyloid | Zendy

Rebekah Mannix | Zendy; Michael J. Whalen | Zendy

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Traumatic Brain Injury, Microglia, and Beta Amyloid

Author(s) -

Rebekah Mannix,

Michael J. Whalen

Publication year - 2012

Publication title -

international journal of alzheimer s disease

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.657

H-Index - 49

eISSN - 2090-8024

pISSN - 2090-0252

DOI - 10.1155/2012/608732

Subject(s) - microglia , traumatic brain injury , inflammation , neuroscience , beta (programming language) , amyloid (mycology) , medicine , disease , amyloid beta , chronic traumatic encephalopathy , pathology , psychology , immunology , poison control , psychiatry , injury prevention , computer science , concussion , programming language , environmental health

Recently, there has been growing interest in the association between traumatic brain injury (TBI) and Alzheimer's Disease (AD). TBI and AD share many pathologic features including chronic inflammation and the accumulation of beta amyloid (A β ). Data from both AD and TBI studies suggest that microglia play a central role in A β accumulation after TBI. This paper focuses on the current research on the role of microglia response to A β after TBI.

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