Preeclampsia, Hypoxia, Thrombosis, and Inflammation | Zendy

Amir A. Shamshirsaz | Zendy; Michael J. Paidas | Zendy; Graciela Krikun | Zendy

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Preeclampsia, Hypoxia, Thrombosis, and Inflammation

Author(s) -

Amir A. Shamshirsaz,

Michael J. Paidas,

Graciela Krikun

Publication year - 2011

Publication title -

journal of pregnancy

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.828

H-Index - 32

eISSN - 2090-2735

pISSN - 2090-2727

DOI - 10.1155/2012/374047

Subject(s) - preeclampsia , medicine , inflammation , hypoxia (environmental) , intrauterine growth restriction , thrombosis , pregnancy , endothelial dysfunction , immunology , cardiology , gestation , biology , genetics , chemistry , organic chemistry , oxygen

Reductions in uteroplacental flow initiate a cascade of molecular effects leading to hypoxia, thrombosis, inflammation, and endothelial cell dysfunction resulting in untoward pregnancy outcomes. In this review, we detail these effects and their relationship to preeclampsia (PE) and intrauterine growth restriction (IUGR).

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