Oxidative Stress, Intrauterine Growth Restriction, and Developmental Programming of Type 2 Diabetes | Zendy

Cetewayo S. Rashid | Zendy; Amita Bansal | Zendy; Rebecca A. Simmons | Zendy

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Oxidative Stress, Intrauterine Growth Restriction, and Developmental Programming of Type 2 Diabetes

Author(s) -

Cetewayo S. Rashid,

Amita Bansal,

Rebecca A. Simmons

Publication year - 2018

Publication title -

physiology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.14

H-Index - 125

eISSN - 1548-9213

pISSN - 1548-9221

DOI - 10.1152/physiol.00023.2018

Subject(s) - intrauterine growth restriction , oxidative stress , endocrinology , medicine , diabetes mellitus , type 2 diabetes , etiology , mitochondrion , oxidative phosphorylation , type 1 diabetes , biology , pregnancy , fetus , genetics , biochemistry

Intrauterine growth restriction (IUGR) leads to reduced birth weight and the development of metabolic diseases such as Type 2 diabetes in adulthood. Mitochondria dysfunction and oxidative stress are commonly found in key tissues (pancreatic islets, liver, and skeletal muscle) of IUGR individuals. In this review, we explore the role of oxidative stress in IUGR-associated diabetes etiology.

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