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Patients with type 1 diabetes demonstrated slower oxygen uptake (V̇o 2 ) kinetics compared with healthy control subjects. Furthermore, a prior bout of high-intensity exercise speeded V̇o 2  kinetics and increased critical power in people with type 1 diabetes. Prior exercise speeded muscle deoxygenation kinetics, indicating that V̇o 2  kinetics in type 1 diabetes are limited primarily by oxygen extraction and/or intracellular factors. These findings highlight the potential for interventions that decrease metabolic inertia for enhancing exercise tolerance in this condition.

Limitations to exercise tolerance in type 1 diabetes: the role of pulmonary oxygen uptake kinetics and priming exercise