The double-edged sword of intermittent hypoxia—can intermittent hypoxia be both deleterious and protective in OSA? Focus on “Frequency and magnitude of intermittent hypoxia modulate endothelial wound healing in a cell culture model of sleep apnea” | Zendy

Lena Lavie | Zendy; Peretz Lavie | Zendy

Open Access

The double-edged sword of intermittent hypoxia—can intermittent hypoxia be both deleterious and protective in OSA? Focus on “Frequency and magnitude of intermittent hypoxia modulate endothelial wound healing in a cell culture model of sleep apnea”

Author(s) -

Lena Lavie,

Peretz Lavie

Publication year - 2017

Publication title -

journal of applied physiology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.253

H-Index - 229

eISSN - 8750-7587

pISSN - 1522-1601

DOI - 10.1152/japplphysiol.00630.2017

Subject(s) - intermittent hypoxia , hypoxia (environmental) , obstructive sleep apnea , sleep apnea , medicine , endothelial dysfunction , inflammation , wound healing , systemic inflammation , bioinformatics , cardiology , immunology , biology , chemistry , organic chemistry , oxygen

whether intermittent hypoxia (IH)—the hallmark of obstructive sleep apnea (OSA)—is detrimental, protective, or both, is a fundamental question. The literature associating OSA with cardiovascular and other comorbidities is overwhelming, yet it is not clear whether IH also exerts protective

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