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fibrosis in vital organs causes significant morbidity and mortality worldwide. Although our understanding of the cellular and molecular mechanisms underlying fibrosis has grown tremendously over the last decades, effective treatments that halt, reverse, or prevent the pathological accumulation and

american journal of physiology. cell physiology

<i>Keeping fibroblasts in suspense: TAZ-mediated signaling activates a context-dependent profibrotic phenotype</i>. Focus on “TAZ activation drives fibroblast spheroid growth, expression of profibrotic paracrine signals, and context-dependent ECM gene expression”

Keeping fibroblasts in suspense: TAZ-mediated signaling activates a context-dependent profibrotic phenotype. Focus on “TAZ activation drives fibroblast spheroid growth, expression of profibrotic paracrine signals, and context-dependent ECM gene expression”