Inactivation of Cyclic AMP Response Element Transcription Caused by Constitutive p38 Activation Is Mediated by Hyperphosphorylation-Dependent CRTC2 Nucleocytoplasmic Transport | Zendy

Huabin Ma | Zendy; Zeyuan Liu | Zendy; ChuanQi Zhong | Zendy; Yifei Liu | Zendy; Zhirong Zhang | Zendy; Yaoji Liang | Zendy; Jingxian Li | Zendy; Shoufa Han | Zendy; Jiahuai Han | Zendy

Open Access

Inactivation of Cyclic AMP Response Element Transcription Caused by Constitutive p38 Activation Is Mediated by Hyperphosphorylation-Dependent CRTC2 Nucleocytoplasmic Transport

Author(s) -

Huabin Ma,

Zeyuan Liu,

ChuanQi Zhong,

Yifei Liu,

Zhirong Zhang,

Yaoji Liang,

Jingxian Li,

Shoufa Han,

Jiahuai Han

Publication year - 2019

Publication title -

molecular and cellular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.14

H-Index - 327

eISSN - 1067-8824

pISSN - 0270-7306

DOI - 10.1128/mcb.00554-18

Subject(s) - phosphoserine , hyperphosphorylation , microbiology and biotechnology , biology , transcription factor , phosphorylation , cytosol , kinase , biochemistry , serine , enzyme , gene

The p38 signal transduction pathway can be activated transiently or constitutively, depending on the contexts in which the activation occurs. However, the biological consequence of constitutive activation of p38 is largely unknown.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research