fpr , a Deficient Xer Recombination Site from a Salmonella Plasmid, Fails To Confer Stability by Dimer Resolution: Comparative Studies with the pJHCMW1 mwr Site | Zendy

Tung Tran | Zendy; David J. Sherratt | Zendy; Marcelo E. Tolmasky | Zendy

Open Access

fpr , a Deficient Xer Recombination Site from a Salmonella Plasmid, Fails To Confer Stability by Dimer Resolution: Comparative Studies with the pJHCMW1 mwr Site

Author(s) -

Tung Tran,

David J. Sherratt,

Marcelo E. Tolmasky

Publication year - 2009

Publication title -

journal of bacteriology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.652

H-Index - 246

eISSN - 1067-8832

pISSN - 0021-9193

DOI - 10.1128/jb.01082-09

Subject(s) - biology , recombination , site specific recombination , plasmid , transposable element , mutagenesis , a site , genetics , binding site , salmonella , sos response , escherichia coli , mutation , dna , mutant , recombinase , gene , bacteria

Salmonella plasmid pFPTB1 includes a Tn3-like transposon and a Xer recombination site, fpr, which mediates site-specific recombination at efficiencies lower than those required for stabilizing a plasmid by dimer resolution. Mutagenesis and comparative studies with mwr, a site closely related to fpr, indicate that there is an interdependence of the sequences in the XerC binding region and the central region in Xer site-specific recombination sites.

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