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Colistin is a drug of last resort for the treatment of many multidrug-resistant Gram-negative bacteria, including carbapenem-resistantKlebsiella pneumoniae (CRKP). However, bacteria readily acquire resistance to this antibiotic via lipopolysaccharide modifications caused by spontaneous mutations or from enzymes acquired by lateral gene transfer. The fitness cost associated with these modifications remains poorly understood.

Colistin Resistance-Mediated Bacterial Surface Modification Sensitizes Phage Infection