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The relevance of apoptosis to killing of intracellular Mycobacterium tuberculosis was studied with phorbol-differentiated THP-1 cells. Microparticles containing isoniazid and rifabutin induced intrinsic apoptosis and bacterial killing equivalent to that with dissolved drugs and maximally enhanced purinergic P2 receptor activity, while drug-free microparticles induced apoptosis via a different mechanism without killing bacteria.

antimicrobial agents and chemotherapy

Enhancement of Apoptosis of THP-1 Cells Infected with<i>Mycobacterium tuberculosis</i>by Inhalable Microparticles and Relevance to Bactericidal Activity

Enhancement of Apoptosis of THP-1 Cells Infected withMycobacterium tuberculosisby Inhalable Microparticles and Relevance to Bactericidal Activity