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TLR13 Recognizes Bacterial 23 S rRNA Devoid of Erythromycin Resistance–Forming Modification
Author(s) -
Marina Oldenburg,
Anne Krüger,
Ruth Ferstl,
Andreas M. Kaufmann,
Gernot Nees,
Anna M. Sigmund,
Barbara Bathke,
Henning Lauterbach,
Mark Suter,
Stefan Dreher,
Uwe Koedel,
Shizuo Akira,
Taro Kawai,
Jan Buer,
Hermann Wagner,
Stefan Bauer,
Hubertus Hochrein,
Carsten J. Kirschning
Publication year - 2012
Publication title -
science
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 12.556
H-Index - 1186
eISSN - 1095-9203
pISSN - 0036-8075
DOI - 10.1126/science.1220363
Subject(s) - erythromycin , microbiology and biotechnology , resistance (ecology) , chemistry , biology , antibiotics , ecology
Host protection from infection relies on the recognition of pathogens by innate pattern-recognition receptors such as Toll-like receptors (TLRs). Here, we show that the orphan receptor TLR13 in mice recognizes a conserved 23S ribosomal RNA (rRNA) sequence that is the binding site of macrolide, lincosamide, and streptogramin group (MLS) antibiotics (including erythromycin) in bacteria. Notably, 23S rRNA from clinical isolates of erythromycin-resistant Staphylococcus aureus and synthetic oligoribonucleotides carrying methylated adenosine or a guanosine mimicking a MLS resistance-causing modification failed to stimulate TLR13. Thus, our results reveal both a natural TLR13 ligand and specific mechanisms of antibiotic resistance as potent bacterial immune evasion strategy, avoiding recognition via TLR13.

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