Open AccessPGE 2 production at sites of tissue injury promotes an anti-inflammatory neutrophil phenotype and determines the outcome of inflammation resolution in vivo
Author(s) -
Catherine A. Loynes,
Jou A. Lee,
Anne L. Robertson,
Michael JG. Steel,
Felix Ellett,
Yi Feng,
Bruce D. Levy,
Moira K. B. Whyte,
Stephen A. Renshaw
Publication year - 2018
Publication title -
science advances
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 5.928
H-Index - 146
ISSN - 2375-2548
DOI - 10.1126/sciadv.aar8320
Subject(s) - efferocytosis , inflammation , lipid signaling , eicosanoid , lipoxin , immunology , in vivo , zebrafish , biology , prostaglandin e , macrophage , microbiology and biotechnology , in vitro , endocrinology , arachidonic acid , biochemistry , gene , enzyme
Neutrophil removal from inflammatory sites is regulated by lipid mediator signals between macrophages and neutrophils.
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