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Two flavonoid-deficient mutants, designated as  fldL-1  and  fldL-2 , were isolated in EMS-mutagenized (0.15%, 10 h) M 2  progeny of grass pea ( Lathyrus sativus  L.). Both the mutants contained total leaf flavonoid content only 20% of their mother varieties. Genetic analysis revealed monogenic recessive inheritance of the trait, controlled by two different nonallelic loci. The two mutants differed significantly in banding patterns of leaf aconitase (ACO) and S-nitrosoglutathione reductase (GSNOR) isozymes, possessing unique bands in  Aco 1, Aco 2,  and  Gsnor 2  loci. Isozyme loci inherited monogenically showing codominant expression in F 2  (1 : 2 : 1) and backcross (1 : 1) segregations. Linkage studies and primary trisomic analysis mapped  Aco 1  and  fld 1  loci on extra chromosome of trisomic-I and  Aco 2 ,  fld 2,  and  Gsnor 2  on extra chromosome of trisomic-IV in linked associations.

the scientific world journal/thescientificworldjournal

Flavonoid-Deficient Mutants in Grass Pea (<i>Lathyrus sativus</i>L.): Genetic Control, Linkage Relationships, and Mapping with Aconitase and S-Nitrosoglutathione Reductase Isozyme Loci

Flavonoid-Deficient Mutants in Grass Pea (Lathyrus sativusL.): Genetic Control, Linkage Relationships, and Mapping with Aconitase and S-Nitrosoglutathione Reductase Isozyme Loci