Homeostatic plasticity mechanisms in mouse V1 | Zendy

Megumi Kaneko | Zendy; Michael P. Stryker | Zendy

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Homeostatic plasticity mechanisms in mouse V1

Author(s) -

Megumi Kaneko,

Michael P. Stryker

Publication year - 2017

Publication title -

philosophical transactions of the royal society b biological sciences

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.753

H-Index - 272

eISSN - 1471-2970

pISSN - 0962-8436

DOI - 10.1098/rstb.2016.0504

Subject(s) - homeostatic plasticity , neuroscience , hebbian theory , visual cortex , plasticity , ocular dominance , neuroplasticity , biology , synaptic scaling , homeostasis , metaplasticity , synaptic plasticity , computer science , microbiology and biotechnology , receptor , physics , artificial neural network , biochemistry , machine learning , thermodynamics

Mechanisms thought of as homeostatic must exist to maintain neuronal activity in the brain within the dynamic range in which neurons can signal. Several distinct mechanisms have been demonstrated experimentally. Three mechanisms that act to restore levels of activity in the primary visual cortex of mice after occlusion and restoration of vision in one eye, which give rise to the phenomenon of ocular dominance plasticity, are discussed. The existence of different mechanisms raises the issue of how these mechanisms operate together to converge on the same set points of activity.This article is part of the themed issue 'Integrating Hebbian and homeostatic plasticity'.

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