Upregulation of IL-1β, IL-6, and CCL-2 by a Novel Mouse Model of Pancreatic Ischemia-Reperfusion Injury | Zendy

Keri E. Lunsford | Zendy; Brandon J. Baird | Zendy; Gregory D. Sempowski | Zendy; Diana M. Cardona | Zendy; Zhiguo Li | Zendy; Kent J. Weinhold | Zendy; Debra L. Sudan | Zendy; Todd V. Brennan | Zendy

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Upregulation of IL-1β, IL-6, and CCL-2 by a Novel Mouse Model of Pancreatic Ischemia-Reperfusion Injury

Author(s) -

Keri E. Lunsford,

Brandon J. Baird,

Gregory D. Sempowski,

Diana M. Cardona,

Zhiguo Li,

Kent J. Weinhold,

Debra L. Sudan,

Todd V. Brennan

Publication year - 2013

Publication title -

transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.45

H-Index - 204

eISSN - 1534-6080

pISSN - 0041-1337

DOI - 10.1097/tp.0b013e318286483a

Subject(s) - chemokine , proinflammatory cytokine , downregulation and upregulation , inflammation , medicine , cytokine , pancreas , reperfusion injury , immunology , endocrinology , ischemia , biology , biochemistry , gene

Little is known about the immunologic events surrounding pancreatic ischemia-reperfusion injury (IRI) because of a lack of established experimental models. The purpose of this study was to develop a mouse model for pancreatic IRI to serve as a basis for the immunologic characterization of pancreatic organ damage at transplantation.

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