Type I Interferon Pathway Mediates Renal Ischemia/Reperfusion Injury | Zendy

Maria Cecília S. Freitas | Zendy; Yoichiro Uchida | Zendy; Charles Lassman | Zendy; Gabriel M. Danovitch | Zendy; Ronald W. Busuttil | Zendy; Jerzy W. KupiecWeglinski | Zendy

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Type I Interferon Pathway Mediates Renal Ischemia/Reperfusion Injury

Author(s) -

Maria Cecília S. Freitas,

Yoichiro Uchida,

Charles Lassman,

Gabriel M. Danovitch,

Ronald W. Busuttil,

Jerzy W. KupiecWeglinski

Publication year - 2011

Publication title -

transplantation

Language(s) - Uncategorized

Resource type - Journals

SCImago Journal Rank - 1.45

H-Index - 204

eISSN - 1534-6080

pISSN - 0041-1337

DOI - 10.1097/tp.0b013e318220586e

Subject(s) - kidney , creatinine , blood urea nitrogen , endocrinology , medicine , necrosis , transplantation , reperfusion injury , receptor , acute tubular necrosis , proinflammatory cytokine , ischemia , tumor necrosis factor alpha , inflammation

Ischemia/reperfusion (I/R) injury is a common cause of acute renal failure after kidney transplantation. This study was designed to analyze the role of type I interferon (IFN) signaling downstream of Toll-like receptor 2/Toll-like receptor 4 activation in the mechanism of I/R-triggered kidney damage.

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