Role of viral replication, antiretroviral therapy, and immunodeficiency in HIV-associated atherosclerosis | Zendy

Priscilla Y. Hsue | Zendy; Peter W. Hunt | Zendy; Amanda Schnell | Zendy; S.C. Kalapus | Zendy; Rebecca Hoh | Zendy; Peter Ganz | Zendy; Jeffrey N. Martin | Zendy; Steven G. Deeks | Zendy

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Role of viral replication, antiretroviral therapy, and immunodeficiency in HIV-associated atherosclerosis

Author(s) -

Priscilla Y. Hsue,

Peter W. Hunt,

Amanda Schnell,

S.C. Kalapus,

Rebecca Hoh,

Peter Ganz,

Jeffrey N. Martin,

Steven G. Deeks

Publication year - 2009

Publication title -

aids

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.195

H-Index - 216

eISSN - 1473-5571

pISSN - 0269-9370

DOI - 10.1097/qad.0b013e32832b514b

Subject(s) - viremia , medicine , viral load , immunology , intima media thickness , inflammation , cohort , lentivirus , human immunodeficiency virus (hiv) , viral disease , carotid arteries

HIV-seropositive patients are at higher risk for atherosclerosis than HIV-seronegative persons. This has been variably attributed to antiretroviral drug toxicity, immunodeficiency, and/or HIV-associated inflammation. To evaluate the contributions of these factors to HIV-associated atherosclerosis, we assessed carotid artery intima-media thickness in a diverse cohort of HIV-seronegative and seropositive adults, including a unique group of HIV-infected patients who were untreated, had undetectable viral loads, and had preserved CD4 T-cell counts (HIV controllers).

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