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We previously reported that radicicol (Hsp90 inhibitor) induced a reduction in the renal blood flow and glomerular filtration rate, in part due to a reduction in urinary NO2/NO3 excretion, suggesting that Hsp90 regulates renal vascular tone in physiological conditions. However, there is a lack of information concerning Hsp90α or Hsp90β role on eNOS activity and their association with acute kidney injury (AKI) characterized by an inadequate NO production. This study evaluated the effects of Hsp90α or Hsp90β intra-renal transfection under ischemia/reperfusion (IR) injury.

Intra-renal transfection of heat shock protein 90 alpha or beta (Hsp90 or Hsp90 ) protects against ischemia/reperfusion injury